The E15R point mutation in scorpion toxin Cn2 uncouples its depressant and excitatory activities on human Nav1.6.

The E15R point mutation in scorpion toxin Cn2 uncouples its depressant and excitatory activities on human Nav1.6.

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  • 16 May 2019
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Participating Institutions

The University of Queensland
The University of Sydney
Australian National University
Monash University
Queensland University of Technology
Edith Cowan University
CSIRO

International Partners

Boston Children’s Hospital, Harvard
University of Washington
University of Tokyo
Massachusetts Institute of Technology
University of Illinois at Urbana-Champaign
University of Southampton

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